KAYNAKLAR (Sindirim Sistemi Hastalıkları)

Kaynaklar

1-Wardhana, Datau EA. Recurrent aphthous stomatitis caused by food allergy. Acta Med Indones. 2010 ;42:236-40.

2-Yaşar S, Yaşar B, Abut E, Aşiran Serdar Z. Clinical importance of celiac disease in patients with recurrent aphthous stomatitis. Turk J Gastroenterol. 2012 ;23:14-8.

3-Olszewska M, Sulej J, Kotowski B. Frequency and prognostic value of IgA and IgG endomysial antibodies in recurrent aphthousstomatitis. Acta Derm Venereol. 2006;86:332-4.

4-Cheng J, Malahias T, Brar P, Minaya MT, Green PH. The association between celiac disease, dental enamel defects, and aphthous ulcers in a United States cohort. J Clin Gastroenterol. 2010 ;44:191-4.

5- Bucci P, Carile F, Sangianantoni A,et al. Oral aphthous ulcers and dental enamel defects in children with coeliac disease. Acta Paediatr. 2006;95:203-7.

6-Mäki M, Aine L, Lipsanen V, Koskimies S. Dental enamel defects in first-degree relatives of coeliac disease patients. Lancet. 1991;337(8744):763-4.

7-Besu I, Jankovic L, Magdu IU,et al. Humoral immunity to cow’s milk proteins and gliadin within the etiology of recurrent aphthousulcers? Oral Dis. 2009 ;15:560-4.

8-Besu I, Jankovic L, Konic-Ristic A,et al. The role of specific cow’s milk proteins in the etiology of recurrent aphthous ulcers. J Oral Pathol Med. 2013 ;42:82-8.

9. Karabulut GS, Beşer OF, Erginöz E,et al. The Incidence of Irritable Bowel Syndrome in Children Using the Rome III Criteria and the Effect of Trimebutine Treatment. J Neurogastroenterol Motil. 2013;19:90-3.

10. Keshteli AH, Dehestani B, Daghaghzadeh H, Adibi P. Epidemiological features of irritable bowel syndrome and its subtypes among Iranian adults. Ann Gastroenterol. 2015;28:253-258.

11. De Silva AP, Nandasiri SD, Hewavisenthi J, et al. Subclinical mucosal inflammation in diarrhea-predominant irritable bowel syndrome (IBS) in a tropical setting.  Scand J Gastroenterol. 2012;47:619-24.

12. Panicker R, Arifhodzic N, Al Ahmad M, Ali SA. Association and symptom characteristics of irritable bowel syndrome among bronchial asthmapatients in Kuwait. Ann Thorac Med. 2010;5:37-42.

13. Powell N, Huntley B, Beech T, et al. Increased prevalence of gastrointestinal symptoms in patients with allergic disease. Postgrad Med J. 2007;83:182-6.

14. Jones VA, McLaughlan P, Shorthouse M, Workman E, Hunter JO. Food intolerance: a major factor in the pathogenesis of irritable bowel syndrome. Lancet 1982; 2: 1115±7.

15. Nanda R, James R, Smith H, Dudley CR, Jewell DP. Food intolerance and the irritable bowel syndrome. Gut 1989; 30:1099-104.

16.  Karamanolis G, Tack J. Nutrition and motility disorders. Best Pract Res Clin Gastroenterol.2006;20:485–505.

17. Zuo XL1, Li YQ, Li WJ, et al. Alterations of food antigen-specific serum immunoglobulins G and E antibodies in patients with irritable bowel syndrome and functional dyspepsia. Clin Exp Allergy. 2007;37:823-30.

18. Stierstorfer MB, Sha CT, Sasson M. Food patch testing for irritable bowel syndrome. J Am Acad Dermatol. 2013;68:377-84. Clin Exp Allergy. 2007 ;37:823-30.

19. Cristofori F, Fontana C, Magistà A, Capriati T, Indrio F et al. Increased prevalence of celiac disease among pediatric patients with irritable bowel syndrome: a 6-year prospective cohort study. JAMA Pediatr. 2014 ;168:555-60

20. Shahbazkhani B, Sadeghi A, Malekzadeh R et al. Non-Celiac Gluten Sensitivity Has Narrowed the Spectrum of Irritable Bowel Syndrome: A Double-Blind Randomized Placebo-Controlled Trial. Nutrients. 2015;7:4542-54.

21. Moayyedi P, Soo S, Deeks JJ, et al. WITHDRAWN: Eradication of Helicobacter pylori for non-ulcer dyspepsia. Cochrane Database Syst Rev. 2011;(2):CD002096.

22. Jin X, Li YM. Systematic review and meta-analysis from Chinese literature: the association betweenHelicobacter pylori eradication and improvement of functional dyspepsia. Helicobacter. 2007;12:541-6.

23. Çınar A, Sadıç M, Atılgan Hİ,  et al. Prevalence of Helicobacter Pylori Infection in School and Pre-School Aged Children with C-14 Urea Breath Test and the Association with Familial and Environmental Factors. Mol Imaging Radionucl Ther. 2015;24:66-70.

24. Tkachenko MA, Zhannat NZ, Erman LV, et al. Dramatic changes in the prevalence of Helicobacter pylori infection during childhood: a 10-year follow-up study in Russia. J Pediatr Gastroenterol Nutr. 2007;45:428-32.

25. Gong EJ, Yun SC, Jung HY, et al. Meta-Analysis of First-Line Triple Therapy for Helicobacter pylori Eradication in Korea: Is It Time to Change? J Korean Med Sci. 2014;29:704-13.

26. Ang TL, Fock KM, Song M, Ang D, Kwek AB, Ong J, Tan J, Teo EK, Dhamodaran S. Ten-day triple therapy versus sequential therapy versus concomitant therapy as first line treatment for Helicobacter pylori infection. J Gastroenterol Hepatol. 2015 Jan 16.

27. Walker MM, Salehian SS, Murray CE,et al. Implications of eosinophilia in the normal duodenal biopsy – an association with allergy and functionaldyspepsia. Aliment Pharmacol Ther. 2010;31:1229-36.

28. Uzunismail H, Goksel S, Cagatay P, Cengiz M. PPIs and food allergy. Am J Gastroenterol. 2010;105:963-4.

29. Saps M, Lu P, Bonilla S. Cow’s-milk allergy is a risk factor for the development of FGIDs in children. J Pediatr Gastroenterol Nutr. 2011;52:166-9.

30. Schäppi MG, Borrelli O, Knafelz D, et al. Mast cell-nerve interactions in children with functional dyspepsia. J Pediatr Gastroenterol Nutr. 2008;47:472-80.
31. Giangreco E, D’agate C, Barbera C,et al. Prevalence of celiac disease in adult patients with refractory functional dyspepsia: value of routine duodenal biopsy. World J Gastroenterol. 2008;14:6948-53.

32. Pomiecinski F, Yang AC, Navarro-Rodrigues T, Kalil J, Castro FF. Sensitization to foods in gastroesophageal reflux disease and its relation to eosinophils in the esophagus: is it of clinical importance? Ann Allergy Asthma Immunol. 2010;105:359-63.

33.  Yükselen A, Celtik  C. Food allergy in children with refractory gastroesophageal reflux disease. Pediatr Int. 2016 ;58:254-8

34.Usai P, Manca R, Cuomo R, et al. Effect of gluten-free diet on preventing recurrence of gastroesophageal reflux disease-related symptoms in adult celiac patients with nonerosive reflux disease. J Gastroenterol Hepatol. 2008;23:1368-72

35. Collin P, Mustalahti K, Kyrönpalo S,et al. Should we screen reflux oesophagitis patients for coeliac disease? Eur J Gastroenterol Hepatol. 2004;16:917-20.

36. Barrie A, Mourabet ME, Weyant K, et al. Recurrent blood eosinophilia in ulcerative colitis is associated with severe disease and primary sclerosing cholangitis. Dig Dis Sci. 2013;58:222-8.

37. Wędrychowicz A, Tomasik P, Pieczarkowski S, et al. Clinical value of serum eosinophilic cationic protein assessment in children with inflammatory bowel disease. Arch Med Sci. 2014;10:1142-6.

38. Myrelid P, Dufmats M, Lilja I,et al. Atopic manifestations are more common in patients with Crohn disease than in the general population. Scand J Gastroenterol. 2004;39:731-6.

39. TAYLOR KB, TRUELOVE SC. Circulating antibodies to milk proteins in ulcerative colitis. Br Med J. 1961;2(5257):924-9.

40. WRIGHT R, TRUELOVE SC. CIRCULATING ANTIBODIES TO DIETARY PROTEINS IN ULCERATIVE COLITIS. Br Med J. 1965;2(5454):142-4.

41. Jewell DP, Truelove SC. Circulating antibodies to cow’s milk proteins in ulcerative colitis. Gut. 1972 ;13:796-801.

42. Casella G, D’Incà R, Oliva L,et al. Prevalence of celiac disease in inflammatory bowel diseases: An IG-IBD multicentre study. Dig Liver Dis. 2010;42:175-8.
43.Tursi A, Giorgetti GM, Brandimarte G, Elisei W. High prevalence of celiac disease among patients affected by Crohn’s disease.Inflamm Bowel Dis. 2005;11:662-6.

44. Leeds JS, Höroldt BS, Sidhu R,et al. Is there an association between coeliac disease and inflammatory bowel diseases? A study of relative prevalence in comparison with population controls. Scand J Gastroenterol. 2007;42:1214-20.

45. Oxford EC, Nguyen DD, Sauk J,et al. Impact of coexistent celiac disease on phenotype and natural history of inflammatory bowel diseases. Am J Gastroenterol. 2013;108:1123-9.

46. Plevy S, Silverberg MS, Lockton S,et al. Combined serological, genetic, and inflammatory markers differentiate non-IBD, Crohn’s disease, and ulcerative colitis patients. Inflamm Bowel Dis. 2013;19:1139-48.

47. Jones VA, Dickinson RJ, Workman E, et al. Crohn’s disease: maintenance of remission by diet. Lancet. 1985 27;2:177-80.

48. Riordan AM, Hunter JO, Cowan RE, et al. Treatment of active Crohn’s disease by exclusion diet: East Anglian multicentre controlled trial. Lancet. 1993;342:1131-4.

49. Ingle SB, Adgaonkar BD, Ingle CR.  Microscopic colitis: Common cause of unexplained nonbloody diarrhea. World J Gastrointest Pathophysiol. 2014;5:48-53.

50. Maguire AA, Greenson JK, Lauwers GY, et al. Collagenous sprue: a clinicopathologic study of 12 cases. Am J Surg Pathol. 2009;33:1440-9.

51. O’Toole A, Coss A, Holleran G,et al. Microscopic colitis: clinical characteristics, treatment and outcomes in an Irish population. Int J Colorectal Dis. 2014;29:799-803.

52.Levy AM, Yamazaki K, Van Keulen VP,et al. Increased eosinophil infiltration and degranulation in colonic tissue from patients withcollagenous colitis. Am J Gastroenterol. 2001;96:1522-8.

53. Weidenhiller M, Müller S, Schwab D,et al. Microscopic (collagenous and lymphocytic) colitis triggered by food allergy. Gut. 2005;54:312-3.

54. Holstein A, Burmeister J, Plaschke A et al. Autoantibody profiles in microscopic colitis. J Gastroenterol Hepatol. 2006;21:1016-20.

55. Roth B, Gustafsson RJ, Ohlsson B.  Auto-antibodies and their association with clinical findings in women diagnosed with microscopic colitis. PLoS One. 2013;8:e66088.

56. Sainsbury A, Sanders DS, Ford AC. Meta-analysis: Coeliac disease and hypertransaminasaemia. Aliment Pharmacol Ther. 2011 Jul;34:33-40.

57. Casella G, Antonelli E, Di Bella C,et al. Prevalence and causes of abnormal liver function in patients with coeliac disease. Liver Int. 2013;33:1128-31.

58. Volta U. Liver dysfunction in celiac disease. Minerva Med. 2008;99:619-29.

59. Saito M, Obi M, Kimura M. Infantile hepatic dysfunction improved by elimination of cows’ milk formulas. Pediatr Immunol. 2005;16:445-8.

60. Freeman HJ. Hepatic manifestations of celiac disease. Clin Exp Gastroenterol. 2010;3:33-9.

61. Bardella MT, Valenti L, Pagliari C,et al. Searching for coeliac disease in patients with non-alcoholic fatty liver disease. Dig Liver Dis. 2004;36:333-6.

62. Rahimi AR, Daryani NE, Ghofrani H,et al. The prevalence of celiac disease among patients with non-alcoholic fatty liver disease in Iran. Turk J Gastroenterol. 2011 Jun;22:300-4.

63. Volta U. Pathogenesis and clinical significance of liver injury in celiac disease. Clin Rev Allergy Immunol. 2009;36:62-70.

64. Caprai S, Vajro P, Ventura A, et al. Autoimmune liver disease associated with celiac disease in childhood: a multicenter study. Clin Gastroenterol Hepatol. 2008;6:803-6.

65. Di Biase AR, Colecchia A, Scaioli E,et al. Autoimmune liver diseases in a paediatric population with coeliac disease – a 10-year single-centre experience. Aliment Pharmacol Ther. 2010;31:253-60.

66. Nastasio S, Sciveres M, Riva S,et al. Celiac disease-associated autoimmune hepatitis in childhood: long-term response to treatment. J Pediatr Gastroenterol Nutr. 2013;56:671-4.

67. Landi A, Weismuller TJ, Lankisch TO, et al. Differential serum levels of eosinophilic eotaxins in primary sclerosing cholangitis, primary biliary cirrhosis, and autoimmune hepatitis. J Interferon Cytokine Res. 2014;34:204-14.

68. Mounajjed T, Oxentenko A, Shmidt E, Smyrk T. The liver in celiac disease: clinical manifestations, histologic features, and response to gluten-free diet in 30 patients. Am J Clin Pathol. 2011 ;136:128-37.

69. Fagoonee S, De Luca L, De Angelis C,et al. Anti-Saccharomyces cerevisiae as unusual antibodies in autoimmune hepatitis. Minerva Gastroenterol Dietol. 2009;55:37-40.

70. Wang JY, Lee CY, Pan PJ, et al. Herb-induced autoimmune-like hepatitis in C57BL/6J mice. Liver Int. 2014;34:583-93.

71. Prandota J, Stolarczyk J. Autoimmune hepatitis associated with the odour of fish food proteins: a causal relationship or just a mere association? Allergol Immunopathol (Madr). 2002;30:331-7.

72. Kokubun M, Kuroda M, Takagi T et al. Eosinophilia in primary biliary cirrhosis. Nihon Shokakibyo Gakkai Zasshi. 1990;87:1410-6.

73. Terasaki S, Nakanuma Y, Yamazaki M, Unoura M. Eosinophilic infiltration of the liver in primary biliary cirrhosis: a morphological study.Hepatology. 1993;17:206-12.

74. Nakamura A, Yamazaki K, Suzuki K, Sato S. Increased portal tract infiltration of mast cells and eosinophils in primary biliary cirrhosis. Am J Gastroenterol. 1997;92:2245-9.

75. Wirth HP, Meyenberger C, Altorfer J, Ammann R, Blum HE. [Eosinophilia in primary biliary cirrhosis: regression under therapy with ursodeoxycholic acid]. Schweiz Med Wochenschr. 1994;124:810-5.

76. Yamazaki K, Suzuki K, Nakamura A, et al. Ursodeoxycholic acid inhibits eosinophil degranulation in patients with primary biliary cirrhosis. Hepatology. 1999;30:71-8.

77.Habior A, Lewartowska A, Orłowska J,et al. Association ofcoeliac disease with primary biliary cirrhosis in Poland.Eur J Gastroenterol Hepatol. 2003;15:159-64.

78. Elinav E, Pinsker G, Safadi R, et al. Association between consumption of Herbalife nutritional supplements and acute hepatotoxicity. J Hepatol. 2007;47:514-20 .

79. Hu C, Deng C, Zhang S,et al. Clinical significance and prevalence of anti-Saccharomyces cerevisiae antibody in Chinese patients with primary biliary cirrhosis. Clin Exp Med. 2013 ;13:245-50.

80. Watanabe H, Ohira H, Kuroda M, et al. Primary sclerosing cholangitis with marked eosinophilic infiltration in the liver. J Gastroenterol. 1995;30:524-8.

81. Ishii M, Iwai M, Harada Y,et al. A role of mast cells for hepatic fibrosis in primary sclerosing cholangitis. Hepatol Res. 2005;31:127-31.

82. Hay JE, Wiesner RH, Shorter RG, LaRusso NF, Baldus WP. Primary sclerosing cholangitis and celiac disease. A novel association. Ann Intern Med. 1988;109:713-7.

83. Cadahía V, Rodrigo L, Fuentes D,et al. Celiac disease (CD), ulcerative colitis (UC), and primary sclerosing cholangitis (PSC) in one patient: a family study. Rev Esp Enferm Dig. 2005;97:907-13.

84. Al-Hussaini A, Basheer A, Czaja AJ. Liver failure unmasks celiac disease in a child. Ann Hepatol. 2013;12:501-5.

85. Granito A, Zauli D, Muratori P, et al. Anti-Saccharomyces cerevisiae and perinuclear anti-neutrophil cytoplasmic antibodies incoeliac disease before and after gluten-free diet. Aliment Pharmacol Ther. 2005;21:881-7.

86. Muratori P, Muratori L, Guidi M, et al. Anti-Saccharomyces cerevisiae antibodies (ASCA) and autoimmune liver diseases. Clin Exp Immunol. 2003;132:473-6.

87. Graham RP, Smyrk TC, Chari ST, Takahashi N, Zhang L. Isolated IgG4-related sclerosing cholangitis: a report of 9 cases. Hum Pathol. 2014 Apr 24. pii: S0046-8177(14)00159-2

88. Yazici A, Orge Gonullu E, Kardes B, Cefle A. The prevalence of atopy in patients with familial Mediterranean fever and Behçet’s disease. Clin Exp Rheumatol. 2013;31(3 Suppl 77):68-70.

89. Torrent Vernetta A, Segarra Cantón O, Soler Palacín P, Segura Cardona RM, Infante Pina D. [Macroamylasaemia in paediatrics].An Pediatr (Barc). 2008;69:439-41.

90. La Villa G, Pantaleo P, Tarquini R,et al. Multiple immune disorders in unrecognized celiac disease: a case report. World J Gastroenterol. 2003;9:1377-80.
91. Bermejo JF, Carbone J, Rodriguez JJ, et al. Macroamylasaemia, IgA hypergammaglobulinaemia and autoimmunity in a patient with Down syndrome and coeliac disease. Scand J Gastroenterol. 2003;38:445-7.

92. Barera G, Bazzigaluppi E, Viscardi M,et al. Macroamylasemia attributable to gluten-related amylase autoantibodies: a case report. Pediatrics. 2001;107:E93.

93. O’Reilly DA, Malde DJ, Duncan T, Rao M, Filobbos R. Review of the diagnosis, classification and management of autoimmune pancreatitis. World J Gastrointest Pathophysiol. 2014;5:71-81.

94. Wang Q, Lu CM, Guo T, Qian JM. Eosinophilia associated with chronic pancreatitis. Pancreas. 2009;38:149-53.

95. Sah RP, Pannala R, Zhang L, et al. Eosinophilia and allergic disorders in autoimmune pancreatitis. Am J Gastroenterol. 2010;105:2485-91

96. Kamisawa T, Anjiki H, Egawa N, Kubota N. Allergic manifestations in autoimmune pancreatitis. Eur J Gastroenterol Hepatol. 2009 ;21:1136-39.

97. Hirano K, Tada M, Isayama H, et al. Clinical analysis of high serum IgE in autoimmune pancreatitis. World J Gastroenterol. 2010;16:5241-6.

98. Masoodi I, Wani H, Alsayari K,et al. Celiac disease and autoimmune pancreatitis: an uncommon association. A case report. Eur J Gastroenterol Hepatol. 2011;23 :1270-2.

99. Kang Nyeong Lee  and Oh Young Lee. The Role of Mast Cells in Irritable Bowel Syndrome. Gastroenterol Res Pract. 2016; 2016: 2031480.

100. Atarodi S, Rafieian S, Whorwell PJ. Faecal incontinence-the hidden scourge of irritable bowel syndrome: a cross-sectional study. BMJ Open Gastroenterol. 2015 Feb 6;1(1):e000002.

Hülya Uzunismail hakkında 210 makale
43 yıl aktif olarak çalışmış, bunun yaklaşık son 25 yılını da, gıda-hastalık ilişkisini bulma uğraşına adamış bir iç hastalıkları ve gastroenteroloji uzmanı olarak derlediğim bilgi ve deneyimlerimi, hem doktorlar hem de hastalar için bir kitapta birleştirerek paylaşmak istedim.